Uniwersytet Warszawski - Centralny System Uwierzytelniania
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Psychology and Neurobiology of Depression

Informacje ogólne

Kod przedmiotu: 2500-EN-PS-EAc1-09
Kod Erasmus / ISCED: 14.4 Kod klasyfikacyjny przedmiotu składa się z trzech do pięciu cyfr, przy czym trzy pierwsze oznaczają klasyfikację dziedziny wg. Listy kodów dziedzin obowiązującej w programie Socrates/Erasmus, czwarta (dotąd na ogół 0) – ewentualne uszczegółowienie informacji o dyscyplinie, piąta – stopień zaawansowania przedmiotu ustalony na podstawie roku studiów, dla którego przedmiot jest przeznaczony. / (0313) Psychologia Kod ISCED - Międzynarodowa Standardowa Klasyfikacja Kształcenia (International Standard Classification of Education) została opracowana przez UNESCO.
Nazwa przedmiotu: Psychology and Neurobiology of Depression
Jednostka: Wydział Psychologii
Grupy: Academic basket
Clinical Psychology basket
Elective courses
electives for 4 and 5 year
Punkty ECTS i inne: 4.00 Podstawowe informacje o zasadach przyporządkowania punktów ECTS:
  • roczny wymiar godzinowy nakładu pracy studenta konieczny do osiągnięcia zakładanych efektów uczenia się dla danego etapu studiów wynosi 1500-1800 h, co odpowiada 60 ECTS;
  • tygodniowy wymiar godzinowy nakładu pracy studenta wynosi 45 h;
  • 1 punkt ECTS odpowiada 25-30 godzinom pracy studenta potrzebnej do osiągnięcia zakładanych efektów uczenia się;
  • tygodniowy nakład pracy studenta konieczny do osiągnięcia zakładanych efektów uczenia się pozwala uzyskać 1,5 ECTS;
  • nakład pracy potrzebny do zaliczenia przedmiotu, któremu przypisano 3 ECTS, stanowi 10% semestralnego obciążenia studenta.
Język prowadzenia: angielski
Rodzaj przedmiotu:

fakultatywne

Założenia (opisowo):

(tylko po angielsku) Students are expected to have taken a course in Psychopathology/Clinical

Psychology


Students must respect the principles of academic integrity. Cheating and

plagiarism (including copying work from other students, internet or other

sources) are serious violations that are punishable and instructors are

required to report all cases to the administration.

Skrócony opis: (tylko po angielsku)

The aim of this research -oriented course is to familiarize students with recent advances in knowledge on depression.

Efekty uczenia się: (tylko po angielsku)

By the end of the course, students will be able to: recognize and describe symptoms of major depression and to differentiate it from other mood disorders; understand the role of genetic , environmental and psychological factors in the etiology of depression; define main risk factors for depression; identify and evaluate main treatment modalities in depression, understand the importance of scientific and clinical research in advancement of knowledge of depression and its treatment.

Zajęcia w cyklu "Semestr zimowy 2023/24" (zakończony)

Okres: 2023-10-01 - 2024-01-28
Wybrany podział planu:
Przejdź do planu
Typ zajęć:
Seminarium, 30 godzin więcej informacji
Koordynatorzy: (brak danych)
Prowadzący grup: Eliza Krzanowska
Lista studentów: (nie masz dostępu)
Zaliczenie: Przedmiot - Zaliczenie na ocenę
Seminarium - Zaliczenie na ocenę
Pełny opis: (tylko po angielsku)

Depression is a serious, recurrent and costly mental disorder that is associated with significant suffering, dysfunction, and decreased quality of life. Depression is very common. At least one in seven people will suffer from depression at some point during their lifetime.

There has been tremendous progress in research on depression. Current evidence supports the view that depression is caused by a complex interaction of psychological, genetic, and environmental factors which through their effect on brain neurobiology lead to a low mood and other symptoms of depression.

The course begins with an overview of the descriptive aspects of depression, including its epidemiology, diagnostic criteria, course and outcome. The next part describes the most popular psychological models of depression as well as specific therapies based on these models. This is followed by a review of research on the role of stressful life events on the onset and recurrence of depression. The next, large part of the course reviews recent advances in genetics, neurocircuitry, and neurobiology of depression as well as it describes biologically-based treatments of depression. And finally, the last part of the course describes depression in specific populations of patients, including women and children.

This course is about half theoretical and half clinical. I try to cover the course material in such a way that each theoretical part is followed by its clinical counterpart. For example, a rather heavy theoretical section on the brain circuitry of depression is immediately followed by clinical application of this knowledge to the development of brain stimulation techniques, such as deep brain stimulation, trans-cranial magnetic stimulation, vagus nerve stimulation, and ECT. Students will also watch filmed interviews with patients who have undergone these treatments.

Literatura: (tylko po angielsku)

Textbook (selected chapters) : I.H Gotlib, C.L. Hammen (Eds.).(2014).

Handbook of Depression (3rd. edition). The Guilford Press, New

York/London. [1 copy will be made available in the reading room of the

faculty library]

Students will additionally be required to read the following journal articles:

1. Caspi A, Sugden K, Moffitt TE, Taylor A, Craig IW, Harrington H,

McClay J, Mill J, Martin J, Braithwaite A, Poulton R. Influence of life

stress on depression: moderation by a polymorphism in the 5-HTT

gene.Science 2003; 301(5631): 386-9

2. Boland , RJ, Keller, MB. 2010. Course and outcome of depression. In

Handbook of Depression, ed. IH Gotlib, CL. Hammen pp. 23-39.

New York: Guilford, 2nd ed.

3. Davison K. Historical aspects of mood disorders. Psychiatry

2008;8:47-51

4. Hames JL, Hagan CR, Joiner TE. Interpersonal processes in

Depression. Annu. Rev. Clin. Psychol. 2013; 9:355-77.

5. Juruena MF. Early-life stress and HPA axis trigger recurrent

adulthood depression. Epilepsy and Behavior 2014; 38: 148-159

6. Kessler RC, Bromet, EJ. Epidemiology of depression across cultures.

Annual Review of Public Health 2014; 34: 119-138

7. Kessler RC, Wang, PS. 2010. Epidemiology of depression. In

Handbook of Depression, ed. IH Gotlib, CL. Hammen pp. 5-22. New

York: Guilford, 2nd ed.

8. Kircanski K., Joorman J., Gotlib IH. Cognitive aspects of depression.

Wiley Interdiscip Rev Cogn SCi 2012; 3(3): 301-313

9. Knekt P et al. Quasi-experimental study on the effectiveness of

psychoanalysis, long-term and short-term psychotherapy on

psychiatric symptoms, work ability and functional capacity during a 5-

year follow-up. J Affect Disord 2011 Jul; 132:37.

10. Krishnan V, Nestler EJ. The molecular Neurobiology of depression.

Nature 2008; 455 (7215): 894-902

11. Lob E, Kirschbaum C, Steptoe A. Persistent depressive symptoms,

HPA-axis hyperactivity, and inflammation: the role of cognitive -

affective and somatic symptoms. Mol Psychiatry 2019; 21 August

12. Lohoff FW. Overview of the genetics of major depressive disorder.

Curr Psychiatry Rep 2010; 539-546.

13. Maughan B., Collishaw S, Stringaris, A. Depression in childhood and

adolescence. J Can Acad Child Adolesc Psychiatry 2013; 22

14. Mulinari, S. Monoamine theories of depression: historical impact on

biomedical research. J Hist Neurosci 2012; 21: 366-392.

15. Price JL, Drevets WC. Neurocicuitry of mood disorders.

Neuropsychopharmacology Reviews 2010; 35: 192-216.

16. Wei L, Ge T, Leng Y, Pan Z, Fan J, Cui R. The role of neural

plasticity in depression: from hippocampus to prefrontal cortex.

Neural Plasticity 2017; 17: 1-17.

Lecture

Number

Topic (Chapter # (pp.), (Article #))

1 Course orientation: Course syllabus, course requirements,

reading materials, grading; historical aspects of depression;

diagnosis and course of depressive disorders (Chapter 2,

Chapter 3, Chapter 4, Chapter 8 (pp. 142-144), Chapter 18,

(3)

2 Epidemiology of depression (Chapter 1), (6)

3 Stress and depression (Chapter 12, Chapter 16), (1)

4 Neurobiology of stress: HPA- axis hyperactivity, reduction in

brain plasticity, and brain inflammation (Chapter 10), (5),

(10), (11), (16)

5 Genetics of depression (Chapter 9), (1), (12)

6 Midterm Exam. Cognitive vulnerability to depression

(Chapter 14), (8)

7 Interpersonal processes and vulnerability to depression

(Chapter 15), (4)

8 Other psychological models of depression

9 Psychological treatments of depression (Chapter 25, Chapter

27, Chapter 29), (9)

10 Neurochemistry of depression (14)

11 Pharmacotherapy of depression (Chapter 26)

12 Brain circuitry of depression; (Chapter 11) (10)

13 Brain stimulation in treatment of depression (Chapter 26),

(15)

14 Depression in women (Chapter 19)

15 Depression in children and adolescents ( Chapter 20)

Opisy przedmiotów w USOS i USOSweb są chronione prawem autorskim.
Właścicielem praw autorskich jest Uniwersytet Warszawski.
Krakowskie Przedmieście 26/28
00-927 Warszawa
tel: +48 22 55 20 000 https://uw.edu.pl/
kontakt deklaracja dostępności USOSweb 7.0.3.0 (2024-03-22)